Bahram Alamdary Badlou
PLTs dysfunction is increased during increased mortality and morbidity processes, in vivo however. During COVID-19 pandemic attacks what were the main cause(s) of PLTs dysfunction is also not elucidated completely. In this paper is focused on different kinds of acute and chronically diseases, and consecutively, PLTs dysfunction after COVID-19 variants infection, as main infectious mutants (antigens) as concrete example, on one hand. On the other hand, focusing on the hematologic disorders like heparin induced thrombosis (HIT), PF4 disorders as not infectious disease, to better highlight PLTs (dys)functioning, that are not still clarified entirely.